Natural Substances and Supplements that Lower Cholesterol
Ph.D. Biophysics, University of Illinois, 1968
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[former Professor and Dean, School of Government, Regent University]
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>A fact sheet for primarily natural substances known to lower plasma cholesterol, compiled from scientific papers and internet sources. Some entries in this fact sheet provide tangential information about cholesterol reduction involving non-natural agents or interactions between agents. A few entries discuss non-natural substances used to lower plasma cholesterol.
|Diet for Dyslipidemia||13|
|Dioscorea and Diosgenin||13|
|Essential Fatty Acids (EFA)||14|
|Flax Seed Fiber||18|
|Fractionated Vegetable Oil||18|
|Hawthorne Berry Extract||21|
|Oat Fiber Source||25|
|Olive Oil -
Olive Oil and Cholesterol
Extra Olive Oil Beats Blood Pressure
|Omega-3 Fatty Acids||26|
|RX Meds Deplete Nutrients||27|
|Red Yeast Rice||28|
|Statins Reduce CoQ10 Level||28|
Source material available but not reviewed at this
time includes information on various additional foods and natural food components that lower cholesterol, as well as substances and conditions
implicated in causing high cholesterol and high LDL levels. For information on some of the following see “Sugars that
Heal” by Mondoa and Kitei (Ballantine Books, 2001). Additional foods are mentioned in other sources. All the following reduce cholesterol except
those which as noted cause different effects.
Aloe extracts (see also glyconutritionals, because the active ingredient in aloe is a polymannose, which is an essential glyconutrient)
Fibrin levels in blood, implicated in arterial deposits
Free radicals implicated in cholesterol-induced plaque formation on blood vessel walls
Fructooligosaccharides and inulin
Glyconutritionals (several of the saccharides in this category are listed individually here)
Insulin, promoted by high glycemic index foods, raises LDL levels
Lady's Thistle Extract
N-Acetylneuraminic Acid, an essential glyconutrient
Niacinamide (but see Niacin above)
Omega-3 Concentrate (but see Omega-3 Fatty Acids above in TOC)
Polysaccharide K (contains 5 of the essential glyconutrients required by the body.
Psyllium Seed Husk Fiber
Soybeans (but see Soy above in TOC)
Viruses, bacteria and mycoplasma implicated in hypercholesteremia
Whole Oat Fiber (but see Fiber and see Oat Fiber Source above in TOC)
A factor in serum lipid concentration is frequency of eating. Eating small meals more often reduces cholesterol. The following is copied from the British Medical Journal website at http://bmj.com/index.dtl
BMJ 2001;323:1286 ( 1 December 2001 )
Frequency of eating and concentrations of serum cholesterol in the Norfolk population of
the European prospective investigation into cancer (EPIC-Norfolk): cross sectional study
Silvia M O Titan, visiting research fellow, a Sheila Bingham, deputy director, b Ailsa Welch, research associate, a Robert Luben, research associate, a Suzy Oakes, research associate, a Nicholas Day, MRC professor of epidemiology, a Kay-Tee Khaw, professor of clinical gerontology. a = Institute of Public Health, University of Cambridge, Cambridge CB2 2SR, b = MRC Dunn Human Nutrition Unit, Cambridge CB2 2XY
Correspondence to: K-T Khaw, Clinical Gerontology Unit, University of Cambridge, Addenbrooke's Hospital Box 251, Cambridge CB2 2QQ firstname.lastname@example.org
Objectives: To examine the relation between self reported eating frequency and serum lipid concentrations in a free living population. Design: Cross sectional population based study. Setting: Norfolk, England. Participants: 14 666 men and women aged 45-75 years from the Norfolk cohort of the European prospective investigation into cancer (EPIC-Norfolk). Main outcome measures: Concentrations of blood lipids. Results: Mean concentrations of total cholesterol and low density lipoprotein cholesterol decreased in a continuous relation with increasing daily frequency of eating in men and women. No consistent relation was observed for high density lipoprotein cholesterol, body mass index, waist to hip ratio, or blood pressure. Mean cholesterol concentrations differed by about 0.25 mmol/l between people eating more than six times a day and those eating once or twice daily; this difference was reduced to 0.15 mmol/l after adjustment for possible confounding variables, including age, obesity, cigarette smoking, physical activity, and intake of energy and nutrients (alcohol, fat, fatty acids, protein, and carbohydrate). Conclusions: Concentrations of total cholesterol and low density lipoprotein cholesterol are negatively and consistently associated with frequency of eating in a general population. The effects of eating frequency on lipid concentrations induced in short term trials in animals and human volunteers under controlled laboratory conditions can be observed in a free living general population. We need to consider not just what we eat but how often we eat.
What is already known on this topic -- Studies in animals and small human trials indicate that eating frequency is inversely related to serum lipid concentrations. Few studies have examined this in a free living population under no dietary restrictions
What this study adds -- In a free living population increased eating frequency was negatively and significantly associated with concentrations of total cholesterol and low density lipoprotein cholesterol. This association was still present after adjustment for body mass index, physical activity, cigarette smoking, and dietary intake.
Mean age adjusted cholesterol concentrations differed by
0.25 mmol/l between people eating more than six times a day and those eating
less than twice daily.
END BMJ excerpt